What human outcomes has PF11 demonstrated in these Early Waves?

Last Updated 2013-05-30

Does this pandemic H1N1 virus, PF11, display any characteristics that differ from Seasonal Influenza?

• Large Scale Direct Lytic Activity (Cell Destruction/Pneumonia) without Secondary Infection involvement
• Rapid Deaths 
• Sudden Heart Attacks in 9 to 22 year old male and female victims
• Breathing Failure after overnight minor symptoms (22 year old student)
• Diffuse Alveolar Damage (DAD) and necrotising bronchiolitis (bilateral gas-exchange and air passage insult)
• Diffuse Alveolar Hemorrhage (DAH) with hemoptysis (bleeding from lungs)
• Acute Respiratory Distress Syndrome (ARDS)
• Heart Attack, Stroke and Pulmonary Embolism, 34 year old Athletic Male
• Acute Necrotizing Encephalopathy (7F) Comatose 8hr post ER release, Brain Dead Day 2 [PubMed, pdf]
• Acute Necrotizing Encephalopathy (4F) [pdf] with Sequelae
• Loss of Movement Control (Child clusters, Melbourne, Australia 2011-09)
• Bleeding into the Brain with Loss of Eyesight (5M)
• Lung Destruction requiring Double Lung Transplant (24M)
• Liver Destruction requiring Transplant (5M)
• Acute Kidney Injury (AKI)
• Acute Liver Injury (23F)
• Disseminated Intravascular Coagulation (DIC)
• Sepsis (Blood Infection)
• Multiple Blood Clots
• Multiple Amputations (23F)
• False Negative Testing results in more than Half of tested cases.
• Ability to Transmit and Grow in Summer Temperatures within Humans
• High Relapse Potential after Treatment and after Testing Negative, with recorded Death outcomes
• Long-Term Complications from Organ Damage
• Brain
• Heart
• Blood Vessels
• Kidneys
• Lung

In the rare cases of death after a normal seasonal influenza infection, a secondary infection is generally the cause of expiration. A bacterium will have opportunistically overtaken the damaged lungs. PF11, on the other hand, has demonstrated the singular ability to kill from direct viral-induced lytic activity and the associated Cytokinic Dysregulation, rapidly destroying masses of cells of its own accord without the involvement of secondary infection. You will understand more about this ability when we cover the Influenza Flux topic, the dangerous period when Influenza is transitioning between species, the Hydra Effect and the gene progression details as you read the Genetic Acquisitions discussion.

Rapid deaths within 12-40 hours of first symptoms generally involving partial viral destruction of major organs including bleeding from the lungs are documented across wide geography (US, Europe, South America, Central America, Africa and Asia) in every age category. Brain infection and kidney damage features robustly across all age categories. Very striking are the sudden heart attacks in 6 year old to 22 year old females and males documented across the globe, with cases occurring after TamiFlu treatment or after being released from a healthcare facility based on achievement of a negative rapid test for a proud announcement of “cured” by the attending physician. 

Testing negative is not a firm predictor of recovery.  The relapse potential is very high.

Disseminated Intravascular Coagulation (DIC), a Cytokinic Dyregulatory systemic clotting of the blood with a resultant swing in the opposite direction (thinning the blood) has proven fatal in this pandemic and, though the term is recent, past pandemics have displayed very similar clinical outcomes.  We have direct clinical reports from one suspect fatal PF11 case (unconfirmed due to no testing) involving necrosis at the skin level, creating a progression of black spots across the body prior to death.

Tracking studies have measured that a well-progressed neurological infection of Influenza leaves long-term loss of function in 25% to 50% of the cases.  A retrospective study of California cases admits serious neurological sequelae in 1 of 20 diagnosed cases of pandemic influenza 2009.  pH1N1 has demonstrated the pathology of Disseminated Intravascular Coagulation (DIC).  We postulate that glucose starvation (brain fuel) and the loss of the detoxifying anti-oxidant effects (O₂) from fresh blood may contribute to long-term neurological loss when the blood is clotting in the brain's capillaries. Case studies after the three pandemics of the past century bear evidence of memory loss, cognitive impairment in multiple categories of reasoning and recognition, partial paralysis and accelerated functional degeneration. Many survivors were “never the same again” according to their families and colleagues.

We're concerned that our public health officials appear to have memory loss concerning the documented effects of Pandemic Influenza in this century as they continue to label this present situation as "mild". 

Children don't have heart attacks from mild Influenza.  Mild Influenza does not harden arteries and then liquefy organs.

For additional background on the clinical and epidemiological observational facts concerning Pandemic Influenza H1N1, please refer to the Table of Contents for PF11 Trends & Issues, Mid-Term.

Please visit GeneWurx.com for insight into the latest published studies.